By inhibiting NF-kappa B p65 and p38 MAPK signaling in an in vitro and in vivo model of ALI, GHK-Cu therapy lowered ROS generation, enhanced superoxide dismutase (SOD) activity, and decreased TNF- and IL-6 production. Moreover, in mice with LPS-induced ALI, GHK-Cu...
We use cookies to ensure that we give you the best experience on our website. If you continue to use this site we will assume that you are happy with it.AcceptPrivacy policy
